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Identification of an evolutionary conserved structural loop that is required for the enzymatic and biological function of tryptophan 2,3-dioxygenase | Scientific Reports
The cellular modifier MOAG‐4/SERF drives amyloid formation through charge complementation | The EMBO Journal
Molecular Neurobiology of Ageing
Delaying aging and the aging-associated decline in protein homeostasis by inhibition of tryptophan degradation | PNAS
Neurodegenerative diseases: Lessons from genome‐wide screens in small model organisms | EMBO Molecular Medicine
Molecular Neurobiology of Ageing
Frontiers | Membrane Interactions and Toxicity by Misfolded Protein Oligomers | Cell and Developmental Biology
Frontiers | Regulation of Age-Related Protein Toxicity | Cell and Developmental Biology
Staff members with discipline Biochemistry & Molecular Biology | Find an expert | How to find us | University of Groningen
The cellular modifier MOAG‐4/SERF drives amyloid formation through charge complementation | The EMBO Journal
Age‐ and disease‐specific changes of the kynurenine pathway in Parkinson's and Alzheimer's disease - Sorgdrager - 2019 - Journal of Neurochemistry - Wiley Online Library
Biology | Free Full-Text | Crosstalk between Different DNA Repair Pathways Contributes to Neurodegenerative Diseases | HTML
Frontiers | N6 -Methyladenosine Negatively Regulates Human Respiratory Syncytial Virus Replication | Cell and Developmental Biology
Identification of MOAG-4/SERF as a Regulator of Age-Related Proteotoxicity - ScienceDirect
Identification of MOAG-4/SERF as a Regulator of Age-Related Proteotoxicity: Cell
Frontiers | Regulation of Age-Related Protein Toxicity | Cell and Developmental Biology
The effectiveness of user-focused mobile health applications in paediatric chronic disease management: A systematic review - Journal of Pediatric Nursing: Nursing Care of Children and Families
Molecular Neurobiology of Ageing
Frontiers | Regulation of Age-Related Protein Toxicity | Cell and Developmental Biology
Identification of MOAG-4/SERF as a Regulator of Age-Related Proteotoxicity: Cell
Delaying aging and the aging-associated decline in protein homeostasis by inhibition of tryptophan degradation | PNAS
C. elegans Model Identifies Genetic Modifiers of α-Synuclein Inclusion Formation During Aging